MS appears to be multiple different diseases with different metabolic pathways.  It is possible that other forms of MS result from exposure to a different antigen.  Since MS prevalence has a correlation with consumption of milk, it follows that this correlation should hold with the most common form of MS, RRMS.

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MS causes demyelination (damage to the protective nerve sheath [insulation]) of the nervous system. Demyelination interferes with transmission of nerve signals. Poor nerve transmission disrupts various body functions causing loss of motor control (legs, feet), sensation of touch, coordination, etc. The body can effect some repair (scar tissue) but some nerve damage is permanent. Removal of the causative antigen can only be expected to halt the progression of the disease and will not repair damage that has already occurred to the nervous system. As a result, monitoring of symptoms is not a reliable means of monitoring disease progression after removal of antigen exposure. The best way to determine if disease progression has halted is with magnetic resonance imaging (MRI). MRI can differentiate between scar tissue (previous damage) and active demyelination. The immune system has “memory”. Removal of an antigen from the body may not immediately halt the immune response. It is assumed that immune response will be greatly diminished within 15 months of removing exposure to the antigen (butyrophilin).